Dysautonomia and Titronic Thermography

Dysautonomia - Synopsis
Dysautonomia - Research
Dysautonomia - History
Dysautonomia - Terminology

Dysautonomia is the failure of the autonomic nervous system to adapt to stimuli or stress. Either the response is erratic, inappropriate in intensity, delayed in timing, or disconnected to the proper thermotome.

The failure of the autonomic nervous system has dramatic and devastating outcomes if not addressed and solved quickly. There are immediate consequences and long-term damages that take place when the sympathetic and parasympathetic branches of the autonomic nervous system are imbalanced and out of sync.

Dysautonomia –

Dysautonomia or autonomic dysfunction is a condition in which the autonomic nervous system does not work properly. This may affect the functioning of the heart, bladder, intestines, sweat glands, pupils, and blood vessels. Dysautonomia has many causes, not all of which may be classified as neuropathic. A number of conditions can feature dysautonomia, such as Parkinson’s disease, multiple system atrophy, and dementia with Lewy bodies, Ehlers-Danlos syndromes, autoimmune autonomic ganglionopathy, and autonomic neuropathy, HIV/AIDS, autonomic failure, and postural orthostatic tachycardia syndrome. Wikipedia

Dysautonomia refers to a disorder of autonomic nervous system (ANS) function that generally involves failure of the sympathetic or parasympathetic components of the ANS, but dysautonomia involving excessive or overactive ANS actions also can occur. Dysautonomia can be local, as in reflex sympathetic dystrophy, or generalized, as in pure autonomic failure. It can be acute and reversible, as in Guillain-Barre syndrome, or chronic and progressive.  NIH.gov

Autonomic Nervous System –

Parasympathetic Nervous System –

Sympathetic Nervous System –

Adaptation –

Thermotome –

 

The physiological effects of dysautonomia include erroneous management of the capillary beds that allow for the dissipation of heat from the core of the body. The parasympathetic fibers that affect and give input to these beds are direct from their centers in the cervical and lumbopelvic portions of the spinal cord. The sympathetic fibers originate in the thoracic segments of the spinal cord and enter into the prolific sympathetic chain ganglia.

When there is an imbalance or interference in the functioning of these centers and their control messages, the heat dissipated will immediately be changed depending on the severity and impact of that interference and imbalance.

Normal functioning of the controls for these capillary beds is constantly modifying the flow of blood on a moment-by-moment basis to regulate core temperature. Normal function is dynamic and balanced.

Abnormal control results in a non-adaptive response to stresses or stimuli, and an unbalanced non-dynamic state.

The Titron Thermography Scanners read the temperature bilaterally simultaneously to gather the data on the paraspinal temperature radiated from these capillary beds. The graphs produced by the scan show the temperature of the scanned anatomy, and with some math, show the balance of the temperatures graphed at that time. Coupled and layered with other graphs from other days and before- and after-visit scans, if patterns of non-adaptation appear, it would indicate an abnormal control system and possible dysautonomia.

The regular use of paraspinal thermography, and the study of the results, would lend great aid to identifying and starting care for someone in the slow slide away from health and vitality into the depths of dysautonomia.